Symptom Research
Neuroimmunology Laboratories
Robert Dantzer, Ph.D. | Peter Grace, Ph.D. | Yuan Pan, Ph.D. | Andrew J. Shepherd, Ph.D.
Principal Investigators
- Departments, Labs and Institutes
- Labs
- Neuroimmunology Laboratories
Areas of Research
- Chemotherapy-Induced Neuropathy
- Chemotherapy-Induced Cognitive Impairments
- Chronic Pain
- Fatigue
- Depression
- Neuron-glia Interactions
The Neuroimmunology Laboratories are led by four well-funded, collaborative principal investigators who share space, equipment, and ideas in a highly interactive, international, research-focused environment. The overall aim of these laboratories is to increase our understanding of the pathophysiology of cancer therapy-induced neurobehavioral toxicities, including neuropathic pain, depressed mood, cognitive dysfunction, and fatigue. This newly acquired knowledge can then be applied to the rational design of novel interventions for alleviating symptom burden in patients with cancer.
Please join us for the next Neurobiology Seminar Series lecture
Contributions of Autoantibodies in Lower Back Pain
Speaker: David J. Clark, M.D.
Professor, Department of Anesthesiology and
Vice Chair, Academic Affairs
Stanford University
December 6, 2024 ● 11:00 am - 12:00 pm CST
Zayed Building, Z2.4040 Rooms 2 and 3
Or join us on Zoom
Meeting ID: 853 8938 0073 ● Password: 155935
The Neuroimmunology Laboratories
Pain Research Symposium
COMING IN 2025: The Advances in Translational Science: Pain Management in Cancer Care symposium will connect you with MD Anderson clinical and basic science experts working to end pain for cancer patients and survivors. Engage in poster sessions, talks and patient-caregiver panels to exchange insights and foster collaboration.
SAVE THE DATE: Friday, May 9, 2025Did You Know?
The Neuroimmunology Laboratories are part of the Department of Symptom Research.
The labs extend the department's ongoing descriptive research and interventional trials by incorporating animal models of pain, fatigue, neuropathy, cognitive impairment, and the underlying neuroinflammatory mechanisms.